1. 国家自然科学基金（82170723）：酪氨酸激酶受体(RTK)信号在足细胞身份维持中的作用及机制（2022.01-2025.12）

2. 国家自然科学基金（81970619）：ANGPT2-ITGAV介导的系膜和足细胞cross-talk在足细胞生理病理中的作用和机制（2020.01-    2023.12）

3. 国家自然科学基金（81670653）：新的足细胞损伤基因PAWR的鉴定及其作用机制（2017.01-2020.12）

4. 国家自然科学基金（81370827）：循环线粒体DNA在足细胞损伤中的作用及其机制（2014.01-2017.12）

5. 江苏省重点研发计划项目（社发面上，BE2020698）：基于人类单细胞RNA测序大数据的microRNA药物毒理预测及其临床应用 （2020.07-2023.06）

6. 江苏省自然科学基金（BK20131324）：线粒体DNA激活的TLR9信号通路在肾小球足细胞损伤中的作用及其机制（2014.07-2016.06）

7. 江苏省临床研究中心子课题（BL2012007）：糖尿病肾病研究 （2013.01-2016.12）

8. 江苏省双创人才计划（2011）

1. Zhang S, Wu J, Zhu X, Song H, Ren L, Tang Q, Xu X, Liu C, Zhang J, Hu W, Liu Z*, Shi S*. A novel approach to identify the mechanism of miR-145-5p toxicity to podocytes based on the essential genes targeting analysis. Mol Ther Nucleic Acids. 2021. 20;26:749-759. (*通讯作者)

2. Bao J, Lu Y, She Q, Dou W, Tang R, Xu X, Zhang M, Zhu L, Zhou Q, Li H, Zhou G, Yang Z, Shi S, Liu Z, Zheng C. MicroRNA-30 regulates left ventricular hypertrophy in chronic kidney disease. JCI Insight. 2021. 24;6(10):e138027.

3. Lu Y, Ye Y, Bao W, Yang Q, Wang J, Liu Z, ShiS*. Genome-wide identification of genes essential for podocyte cytoskeletons based on single-cell RNA sequencing. Kidney Int. 2017. 92(5):1119-1129. (*通讯作者)

4. Lang Y, Zhao Y, Zheng C, Lu Y, Wu J, Zhu X, Zhang M, Yang F, Xu X, Shi S*, Liu Z*. MiR-30 family prevents uPAR-ITGB3 signaling activation through calcineurin-NFATC pathway to protect podocytes. Cell Death Dis. 2019. 24;10(6):401. (*通讯作者)

5. Lu Y, Ye Y, Yang Q, Shi S*. Single-cell RNA-sequence analysis of mouse glomerular mesangial cells uncovers mesangial cell essential genes.. Kidney Int. 2017. 92(2):504-513. (*通讯作者)

6. Zhao Y, Wu J, Zhang M, Zhou M, Xu F, Zhu X, Zhou X, Lang Y, Yang F, Yun S, Shi S*, Liu Z*. Angiotensin II induces calcium/calcineurin signaling and podocyte injury by downregulating microRNA-30 family members. J Mol Med (Berl). 2017. 95(8): 887-898. (*通讯作者)

7. Bao W, Xia H, Liang Y, Ye Y, Lu Y, Xu X, Duan A, He J, Chen Z, Wu Y, Wang X, Zheng C, Liu Z*, Shi S*. Toll-like Receptor 9 Can be Activated by Endogenous Mitochondrial DNA to Induce Podocyte Apoptosis. Sci Rep. 2016. 6:22579. (*通讯作者)

8. Wu J, Zheng C, Wang X, Yun S, Zhao Y, Liu L, Lu Y, Ye Y, Zhu X, Zhang C, Shi S*, Liu Z*. MicroRNA-30 family members regulate calcium/calcineurin signaling in podocytes. J Clin Invest. 2015.125(11):4091-106. (*通讯作者)

9. Zhang C, Zhang W, Chen HM, Liu C, Wu J, Shi S*, Liu Z*. Plasma microRNA-186 and proteinuria in focal segmental glomerulosclerosis. Am J Kidney Dis. 2015. 65(2):223-32. (*通讯作者) 

10. Gentle ME*, Shi S*, Daehn I, Zhang T, Qi H, Yu L, D'Agati VD, Schlondorff DO, Bottinger EP. Epithelial cell TGF-β signaling induces acute tubular injury and interstitial inflammation. J Am Soc Nephrol. 2013. 24(5):787-99. (*共同第一）

11. Shi S*, Yu L, Chiu C, Sun Y, Chen J, Khitrov G, Merkenschlager M, Holzman LB, Zhang W, Mundel P, Bottinger EP*. Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis. J Am Soc Nephrol. 2008. 19(11):2159-69. (*通讯作者)

12. Krick S, Shi S, Ju W, Faul C, Tsai SY, Mundel P, Böttinger EP. Mpv17l protects against mitochondrial oxidative stress and apoptosis by activation of Omi/HtrA2 protease. Proc Natl Acad Sci USA. 2008. 105(37):14106-11.

13. Shi S, GeC, Luo Y, Hou X, Haltiwanger RS, Stanley P. The threonine that carries fucose, but not fucose, is required for Cripto to facilitate Nodal signaling. J Biol Chem. 2007. 282(28):20133-41.

14. Shi S, Stahl M, Lu L, Stanley P. Canonical Notch signaling is dispensable for early cell fate specifications in mammals. Mol Cell Biol. 2005. 25(21):9503-8.

15. Shi S, Williams SA, Seppo A, Kurniawan H, Chen W, Ye Z, Marth JD, Stanley P. Inactivation of the Mgat1 gene in oocytes impairs oogenesis, but embryos lacking complex and hybrid N-glycans develop and implant. Mol Cell Biol. 2004. 24(22):9920-9. 

16. Shi S, Stanley P. Protein O-fucosyltransferase 1 is an essential component of Notch signaling pathways. Proc Natl Acad Sci USA. 2003.100(9):5234-9. 

1. 国家科技进步二等奖（2020年）: 肾小球肾炎诊治策略和关键技术的创新与应用（排名：5/10）

2. 江苏省科技进步一等奖（2017年）: 足细胞损伤机制和临床转化研究（排名：4/10）